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Unique Identifier 86052160
Authors: Finucane P. Doyle CT. Ferriss JB. Molloy M. Murnaghan D.
Institution: Zentrum Anaesthesiologie, Rettungs- und Intensivmedizin, Universitat Gottingen.
Title: Behcet's syndrome with myositis and glomerulonephritis.
Source: British Journal of Rheumatology. 24(4):372-5, 1985 Nov.
Abstract: Necrotizing myositis and acute proliferative glomerulonephritis is described in a patient with Behcet's syndrome. Both of these rare manifestations resolved spontaneously while the underlying disease remains active.

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Unique Identifier 85125727
Authors: Vilppula AH. Aine RA.
Institution Cardiac Unit, Massachusetts General Hospital and Harvard Medical School, Boston 02114, USA.
Title: Polymyositis associated with several immunological disorders.
Source: Clinical Rheumatology. 3(4):533-9, 1984 Dec.
Abstract: A case of polymyositis associated with scleroderma, adult coeliac disease, diabetes mellitus and IgA nephropathy is reported in a 33-year-old male.

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Unique Identifier 81103111
Authors: Elling H. Kristensen IB.
Institution Cardiovascular Health Research Unit, Seattle, WA 98101, USA.
Title: Fatal renal failure in polymyalgia rheumatica caused by disseminated giant cell arteritis.
Source: Scandinavian Journal of Rheumatology. 9(4):206-8, 1980.
Abstract: A 62-year-old man with classical signs of polymyalgia rheumatica for one year suddenly developed fatal renal failure, despite treatment with steroids. Autopsy showed disseminated giant cell arteritis in the kidneys, pancreas, oesophagus, and larynx, with the most pronounced changes in the kidneys. All intrarenal arteries and arterioles were affected. The changes ranged from occlusive intimal proliferation to severe fibrinoid necrosis in the whole arterial wall. Fibrinoid necrosis was also found in the afferent and efferent arteries and several thromboses were seen in the glomeruli. It is not known why this apparently non-complicated case of polymyalgia rheumatica suddenly turned into a fatal case of disseminated giant cell arteritis. The only precipitating factor to be incriminated seems to be a reduction in the corticosteroid dose. This case adds further support to suggestions of the existence of a distinct type of systemic giant cell arteritis.

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